Glenda L. Keating, Ph.D.
Research Associate
gkeatin@emory.edu
Phone: 404.727.3305

Education
Dalhousie University, Nova Scotia, Canada B.Sc. 1994 Neuroscience & Biology
University of St. Andrews, St. Andrews, Scotland Ph.D. 1999 Psychology
Emory University, Atlanta, GA, USA Postdoctoral Training 1999-2003 Neurology

Positions and Employment

1994-1995 Research Assistant, Department of Psychology, Dalhousie University, Nova Scotia, Canada.

1995-1999 Graduate Student, Department of Psychology, University of St. Andrews, St. Andrews, Scotland

1999-2003 Postdoctoral Fellow, Department of Neurology, Emory University, Atlanta, GA

2004- Research Associate, Department of Neurology, Emory University, Atlanta, GA

Honors and Awards

1990 Canada Science Scholar Entrance Scholarship- Dalhousie University

1994 Graduated Magnis Cum Honoribus, Dalhousie University

1994 Dalhousie University Dean's List

1995-1998 Overseas Research Studentship (sponsored by the CVCP)

1995-1998 Sir Harold Mitchell Foundation Studentship

1999 American Academy of Sleep Medicine Postdoctoral Fellowship

2000 Applied Physiological and Sleep Sciences Society Merit Award

2000 Restless Legs Syndrome Foundation Fellowship

Grant Support

Ongoing Research Support

R01 HL727221 Decker (PI) 07/01/02-06/31/06

NHLBI
Episodic Neonatal Hypoxia Impairs Sleep and Cognition
The major goals of this study are to characterize neurochemical and behavioral sequellae of intermittent hypoxic insults occurring during the neonatal period.
Role: Co-investigator

R01 NS 43374 Rye (PI) 04/01/02 - 03/31/06

NINDS
Functional Circuitry of Midbrain Dopamine in Sleep and Wake
The major goals of this project are to characterize thalamic and other midbrain dopaminergic substrates and their role in rodent and non-human primate wake/sleep architecture.
Role: Personnel

Completed Research Support

R01 NS 36697 Rye (PI) 12/01/98-11/30/02

NINDS
Brainstem Substrates of Dopamine Modulated Movements

The major goals of this project are to precisely delineate the brainstem targets of basal ganglia output nuclei and the premotor brainstem structures which are innervated in turn. Methods involve electron and light microscopy and functional neuroanatomy. The sleep architectures of animals depleted of striatal dopamine will be compared to those with lesions of brainstem regions indirectly modulated by dopamine.
Role: Personnel

Memberships

1996-present Member, Society for Neuroscience

1998-present Member, American Academy of Sleep Medicine